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Literature summary for 2.1.1.142 extracted from
- The sterol methyltransferases SMT1, SMT2, and SMT3 influence Arabidopsis development through nonbrassinosteroid products (2010), Plant Physiol., 153, 741-756.
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Arabidopsis thaliana | - |
- |
- |
Synonyms
| Synonyms | Comment | Organism |
|---|---|---|
| SMT-2 | - |
Arabidopsis thaliana |
| SMT-3 | - |
Arabidopsis thaliana |
General Information
| General Information | Comment | Organism |
|---|---|---|
| malfunction | Deficiency of SMT2 in the cvp1 mutant results in moderate developmental defects, including aberrant cotyledon vein patterning, serrated floral organs, and reduced stature, but plants are viable, suggesting that SMT3 activity can substitute for the loss of SMT2. The overexpression of SMT3 complements the cotyledon venation pattern defects of cvp1 (SMT2) mutants | Arabidopsis thaliana |
| malfunction | smt3 single mutants appear wild type, cvp1 smt3 double mutants show enhanced defects relative to cvp1 mutants, such as discontinuous cotyledon vein pattern, and produce novel phenotypes, including defective root growth, loss of apical dominance, sterility, and homeotic floral transformations. Antisense smt3 plants did not display a phenotype, suggesting that SMT3 and SMT2 are functionally redundant | Arabidopsis thaliana |
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